Quote:
Originally Posted by Travis
Do you think there is anyway one can actually stimulate HPTA even while on at lower doses...or even higher doses for that matter.
I was actually looking for your feedback on this, thanx for stoppin in!
|
The answer is yes but not using this protocol. However, the real question is why would one do this if they plan to stay on-cycle for an extend period beyond the administration of the stimulation of the HPTA. In doing so, they would simply suppress themselves again.
The general logic for such a protocol has always been "to maintain testicular mass and proper function of the HPTA" but that just does not hold water when the actual outcome is examined.
First, the idea of testicular mass itself is a complete joke. I have seen many many guys who have run some application of Clomid, HCG or whatnot while on cycle to maintain testicular mass. Testicular mass is NOT indicitive of proper HPTA function. In short, you can have "cannonballs" and still be suppressed. It is not about testicular mass.
Second, the protocol the author is describing is...
"...a gram or two of test with arimidex to keep water off with fina 75 to 150mg every other day for 4 weeks --then 2 to 3 weeks of cruising (test at 300-400mg and clomid at 5 (day one), 4(day two), 3(day three),then 2 every day for 2 weeks)--and then back on everything full again (maybe equipoise used instead of fina this time)for 4 weeks (then 2 to 3 weeks cruising again etc etc)
A "gram or two of test" and 75-100 mg of Fina EOD for four weeks will interrupt the HPTA to a point that Clomid will not provide a means of restoration in 2-3 weeks of administration. Period.
Now, one could use HCG as well as Clomid and an AI but this again would only result in suppression if the individual in question continues to use AAS after administration of the HCG, Clomid and AI.
Again, as I stated before in other threads, and to summarize here, there is little value in running PCT-type compounds during long cycles of AAS. The idea of testicular size is not indicitive of a properly functioning HPTA nor, have I ever seen a case where on-cycle administration of PCT-type compounds resulted "lesser suppression" (BTW - that description leaves a lot of discussion as well) after the cycle. In every case, PCT is still a requirement.
Regards,