Novedex XT and Hypertest

[mplante21]

I want to start taking a good tribulus t-booster along with an anti-estrogen supp. How should i go about taking these supplements? This is what I had in mind for 8 weeks.

Hypertest - 4 Caps/day for 8 weeks (4,4,4,4,4,4,4,4)

Novedex XT - (2,3,4,3,2,1,0,0) Obviously I would work my way up and taper it down.

After the 8 weeks of Hypertest and 6 wks of Novedex XT I would take 2 weeks off from Hypertest and 4 wks off from Novedex XT and start up another 8-10 wk cycle of the same supps.

Should I just stick to one or the other? Or will both be fine to do together? Keep in mind this isnt PCT.

What would be the proper way to stack both supps together?

Thanks in advance.

[rev2217]

If you want to run a natty test booster and an AI, I would NOT recomend Novedext...

Your best bet would be a natty test booster and formestane.

TransForm is an excellent formestane option.

...and yes, start your AI with the test booster and taper it down. Or wait a week then start the AI and taper down, running the AI 1 week past the last week of your test booster.

[rev2217]

...I have to add this to my last post, so that you are aware of why any ATD is a bad choice when trying to increase Test...for pct or otherwise.

Why ATD is the WORST choice in PCT! - Science of Muscle Forums

Formestane on the otherhand will help to support test production and utilization!

[jslep]

Originally Posted by rev2217

...I have to add this to my last post, so that you are aware of why any ATD is a bad choice when trying to increase Test...for pct or otherwise.

Why ATD is the WORST choice in PCT! - Science of Muscle Forums

Formestane on the otherhand will help to support test production and utilization!

good food for thought!

[steam]

4 novedext?! say goodbye to your libido!

[Djinn]

I've had great success with ATDs so I disagree. My libido did not lower and I gained strength and mass. AromX by AMS and Inhibit E are the ones I used and both are phenomenal products. Formestane is fine too i'm not knocking it by any means but ATDs work well. I'd go read on JAMA before buying into something on Science of muscle forums.

Cheers,
Ali

[steam]

was that ATD novedext specifically? ...up to OP to give it a whirl but i have read so many peeps cry abt libido loss at near that dosage. YMMV but if it happens, just scale back.

[rev2217]

Originally Posted by Djinn

I've had great success with ATDs so I disagree. My libido did not lower and I gained strength and mass. AromX by AMS and Inhibit E are the ones I used and both are phenomenal products. Formestane is fine too i'm not knocking it by any means but ATDs work well. I'd go read on JAMA before buying into something on Science of muscle forums.

Cheers,
Ali

It's cool to disagree, but SOM is an awesome site with lots of good solid info.

Also...here is a little something from pubmed on the subject...also quoted at SOM...

Effects of ATD on male sexual behavior and androge...[Horm Behav. 1989] - PubMed Result

Effects of ATD on male sexual behavior and androgen receptor binding: a reexamination of the aromatization hypothesis.
Kaplan ME, McGinnis MY.

Department of Anatomy, Mount Sinai School of Medicine, CUNY, New York 10029.

The aromatization hypothesis asserts that testosterone (T) must be aromatized to estradiol (E2) to activate copulatory behavior in the male rat. In support of this hypothesis, the aromatization inhibitor, ATD, has been found to suppress male sexual behavior in T-treated rats. In our experiment, we first replicated this finding by peripherally injecting ATD (15 mg/day) or propylene glycol into T-treated (two 10-mm Silastic capsules) or control castrated male rats. In a second experiment, we bilaterally implanted either ATD-filled or blank cannulae into the medial preoptic area (MPOA) of either T-treated or control castrated male rats. With this more local distribution of ATD, a lesser decline in sexual behavior was found, suggesting that other brain areas are involved in the neurohormonal activation of copulatory behavior in the male rat. To determine whether in vivo ATD interacts with androgen or estrogen receptors, we conducted cell nuclear androgen and estrogen receptor binding assays of hypothalamus, preoptic area, amygdala, and septum following treatment with the combinations of systemic T alone. ATD plus T, ATD alone, and blank control. In all four brain areas binding of T to androgen receptors was significantly decreased in the presence of ATD, suggesting that ATD may act both as an androgen receptor blocker and as an aromatization inhibitor. Competitive binding studies indicated that ATD competes in vitro for cytosol androgen receptors, thus substantiating the in vivo antiandrogenic effects of ATD. Cell nuclear estrogen receptor binding was not significantly increased by exposure to T in the physiological range. No agonistic properties of ATD were observed either behaviorally or biochemically. Thus, an alternative explanation for the inhibitory effects of ATD on male sexual behavior is that ATD prevents T from binding to androgen receptors.

...also I have used ATDs as well, but a non ATD AI is a much better choice...imo

[littlrook]

ATD
-pros, lose water weight, gain strength
-cons, loss of libido, acne for me

Formestane
-pros, lose water weight, gain strength, libido way up
-cons, doesn't dry me out as much as atd

[Djinn]

So okay some SOLID articles here LoL! Thanks acutally I love reading good scientific studies. I guess ATD just doesn't hit me that badly. I'm lucky I guess. It looks like I'm going to discover the wonders of Formestane soon though.

Originally Posted by rev2217

It's cool to disagree, but SOM is an awesome site with lots of good solid info.

Also...here is a little something from pubmed on the subject...also quoted at SOM...

Effects of ATD on male sexual behavior and androge...[Horm Behav. 1989] - PubMed Result

Effects of ATD on male sexual behavior and androgen receptor binding: a reexamination of the aromatization hypothesis.
Kaplan ME, McGinnis MY.

Department of Anatomy, Mount Sinai School of Medicine, CUNY, New York 10029.

The aromatization hypothesis asserts that testosterone (T) must be aromatized to estradiol (E2) to activate copulatory behavior in the male rat. In support of this hypothesis, the aromatization inhibitor, ATD, has been found to suppress male sexual behavior in T-treated rats. In our experiment, we first replicated this finding by peripherally injecting ATD (15 mg/day) or propylene glycol into T-treated (two 10-mm Silastic capsules) or control castrated male rats. In a second experiment, we bilaterally implanted either ATD-filled or blank cannulae into the medial preoptic area (MPOA) of either T-treated or control castrated male rats. With this more local distribution of ATD, a lesser decline in sexual behavior was found, suggesting that other brain areas are involved in the neurohormonal activation of copulatory behavior in the male rat. To determine whether in vivo ATD interacts with androgen or estrogen receptors, we conducted cell nuclear androgen and estrogen receptor binding assays of hypothalamus, preoptic area, amygdala, and septum following treatment with the combinations of systemic T alone. ATD plus T, ATD alone, and blank control. In all four brain areas binding of T to androgen receptors was significantly decreased in the presence of ATD, suggesting that ATD may act both as an androgen receptor blocker and as an aromatization inhibitor. Competitive binding studies indicated that ATD competes in vitro for cytosol androgen receptors, thus substantiating the in vivo antiandrogenic effects of ATD. Cell nuclear estrogen receptor binding was not significantly increased by exposure to T in the physiological range. No agonistic properties of ATD were observed either behaviorally or biochemically. Thus, an alternative explanation for the inhibitory effects of ATD on male sexual behavior is that ATD prevents T from binding to androgen receptors.

...also I have used ATDs as well, but a non ATD AI is a much better choice...imo